The nurse prepares discharge instructions for the client diagnosed with a myocardial infarction. Which client statement indicates a need for further education? "I will take:

A. Wheezes on inspiration: Wheezing is typically associated with obstructive pulmonary conditions, such as asthma or chronic obstructive pulmonary disease (COPD), and is caused by narrowing of the airways. In ARDS, the pathophysiology involves inflammation and fluid accumulation in the alveoli, which leads to impaired oxygen exchange but not typically to wheezing. Instead, crackles or rales (a fine, wet sound) are more commonly heard on auscultation in ARDS, particularly as fluid builds up in the alveoli.

B. Blood pressure 170/90: Although ARDS can be associated with hemodynamic instability, elevated blood pressure (170/90 mmHg) is not a typical finding. In fact, ARDS is more commonly associated with low blood pressure or hypotension, particularly if the client is experiencing shock or is on mechanical ventilation. Elevated blood pressure could suggest another issue, such as pain, anxiety, or the use of medications like vasopressors. It is not directly related to the pulmonary edema seen in ARDS.

C. Tachypnea: Tachypnea, or rapid breathing, is a hallmark clinical manifestation of acute respiratory distress syndrome (ARDS). In ARDS, pulmonary edema (fluid accumulation in the lungs) occurs as a result of damage to the alveolar-capillary membrane, leading to impaired gas exchange. The body attempts to compensate for decreased oxygenation by increasing the respiratory rate, leading to tachypnea. This is an early sign of respiratory distress and often precedes hypoxemia and other more severe manifestations. The nurse should closely monitor for tachypnea, as it can indicate worsening respiratory compromise.

D. Bradycardia: Bradycardia, or a slow heart rate, is not typically associated with ARDS. In fact, tachycardia (an elevated heart rate) is more commonly seen in response to hypoxia, respiratory distress, or as a compensatory mechanism for low blood pressure in critical illness. Bradycardia could indicate other issues such as vagal stimulation, medication effects, or electrolyte imbalances but is not characteristic of ARDS itself. 4o mini

A. Sublingual nitroglycerin each day to prevent chest pain:
The statement about taking sublingual nitroglycerin daily is incorrect. Sublingual nitroglycerin is prescribed as needed for acute chest pain (angina) or for relieving chest discomfort associated with myocardial infarction (MI). It is not intended for daily use as a preventive measure. The client should be educated to use sublingual nitroglycerin only when experiencing chest pain, and if the pain persists or worsens, they should seek immediate medical help. Daily or routine use of nitroglycerin should be avoided unless specifically instructed by a healthcare provider. The client should also be educated about other preventive measures like long-acting nitrates if needed for chest pain prevention.

Explanation of other options:

B. Clopidogrel to prevent clots from forming:
This statement is correct. Clopidogrel (Plavix) is an antiplatelet medication commonly prescribed after a myocardial infarction to help prevent the formation of blood clots, which can reduce the risk of future cardiovascular events such as stroke or further heart attacks. It works by inhibiting platelets from clumping together, which is crucial in the post-MI period.

C. Atorvastatin at bedtime to reduce my cholesterol levels:
This statement is correct. Atorvastatin is a statin medication that is used to lower cholesterol levels, particularly LDL cholesterol (the "bad" cholesterol), which contributes to atherosclerosis and increases the risk of further heart problems. Statins are often prescribed after a myocardial infarction to help reduce the risk of future events. Taking atorvastatin at bedtime is also a common recommendation because cholesterol synthesis is higher during the night.

D. Metoprolol to decrease my heart's need for oxygen:
This statement is correct. Metoprolol is a beta-blocker that reduces the heart rate and blood pressure, which in turn lowers the heart's oxygen demand. After a myocardial infarction, beta-blockers like metoprolol are commonly prescribed to prevent further heart damage, reduce ischemia, and decrease the likelihood of arrhythmias. They help in the management of heart failure as well by improving the heart's efficiency.