The forward effects in a patient with left ventricular heart failure include (Select all that apply)
oliguria.
increased heart rate.
wet cough.
confusion.
jugular venous distention.
Correct Answer : B,C
Choice A reason: Oliguria is not a forward effect of left ventricular heart failure. It is a backward effect that occurs due to reduced renal perfusion and activation of the renin-angiotensin-aldosterone system.
Choice B reason: Increased heart rate is a forward effect of left ventricular heart failure. It is a compensatory mechanism that occurs due to reduced cardiac output and sympathetic stimulation.
Choice C reason: Wet cough is a forward effect of left ventricular heart failure. It is a sign of pulmonary congestion and edema that result from increased pressure in the pulmonary veins and capillaries.
Choice D reason: Confusion is not a forward effect of left ventricular heart failure. It is a backward effect that occurs due to reduced cerebral perfusion and hypoxia.
Choice E reason: Jugular venous distention is not a forward effect of left ventricular heart failure. It is a backward effect that occurs due to increased pressure in the right atrium and vena cava.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A reason: This is not the cause of airway obstruction in asthma. Collapse of the cartilaginous rings in the bronchi is a feature of tracheobronchomalacia, a condition in which the airways are weak and floppy.
Choice B reason: This is not the cause of airway obstruction in asthma. Type II alveolar cell injury and decreased surfactant are associated with acute respiratory distress syndrome (ARDS), a condition in which the alveoli are damaged and filled with fluid.
Choice C reason: This is not the cause of airway obstruction in asthma. Alveolar changes and pulmonary congestion are seen in chronic obstructive pulmonary disease (COPD), a condition in which the alveoli are enlarged and lose their elasticity.
Choice D reason: This is the correct cause of airway obstruction in asthma. Asthma is a chronic inflammatory disorder of the airways, characterized by mucus secretion, bronchoconstriction, and airway edema. These factors reduce the diameter of the airways and increase the resistance to airflow.
Correct Answer is D
Explanation
Choice A reason: The patient should not use the glucocorticoid as needed when symptoms flare. The glucocorticoid is a long-term controller medication that reduces inflammation and prevents exacerbations of COPD. It should be used regularly as prescribed, not as a rescue medication.
Choice B reason: The patient does not need to use the beta2-adrenergic agonist drug daily even when they don't have any symptoms. The beta2-adrenergic agonist is a short-acting bronchodilator that relaxes the smooth muscles of the airways and improves airflow. It should be used as needed for relief of acute symptoms, not as a maintenance medication.
Choice C reason: The beta2-adrenergic agonist does not suppress the synthesis of inflammatory mediators. The beta2-adrenergic agonist is a bronchodilator that acts on the beta2 receptors of the airways and causes relaxation of the smooth muscles. It does not have any anti-inflammatory effects.
Choice D reason: The glucocorticoid is used as prophylaxis to prevent exacerbations every day. This is the correct statement that indicates understanding of this medication regimen. The glucocorticoid is a long-term controller medication that reduces inflammation and prevents exacerbations of COPD. It should be used regularly as prescribed, along with the beta2-adrenergic agonist as needed for relief of acute symptoms.
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