The forward effects in a patient with left ventricular heart failure include (Select all that apply)
oliguria.
increased heart rate.
wet cough.
confusion.
jugular venous distention.
Correct Answer : B,C
Choice A reason: Oliguria is not a forward effect of left ventricular heart failure. It is a backward effect that occurs due to reduced renal perfusion and activation of the renin-angiotensin-aldosterone system.
Choice B reason: Increased heart rate is a forward effect of left ventricular heart failure. It is a compensatory mechanism that occurs due to reduced cardiac output and sympathetic stimulation.
Choice C reason: Wet cough is a forward effect of left ventricular heart failure. It is a sign of pulmonary congestion and edema that result from increased pressure in the pulmonary veins and capillaries.
Choice D reason: Confusion is not a forward effect of left ventricular heart failure. It is a backward effect that occurs due to reduced cerebral perfusion and hypoxia.
Choice E reason: Jugular venous distention is not a forward effect of left ventricular heart failure. It is a backward effect that occurs due to increased pressure in the right atrium and vena cava.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A reason: This is not a physiological response to hydralazine. Cool extremities are a sign of poor peripheral perfusion, which can be caused by vasoconstriction, not vasodilation.
Choice B reason: This is not a physiological response to hydralazine. Increased urinary output is a sign of diuresis, which can be caused by diuretic medications, not vasodilators.
Choice C reason: This is not a physiological response to hydralazine. Pale skin is a sign of reduced blood flow to the skin, which can be caused by vasoconstriction, not vasodilation.
Choice D reason: This is a physiological response to hydralazine. Reflex tachycardia is a compensatory mechanism that occurs when the blood pressure drops due to vasodilation. The heart rate increases to maintain the cardiac output and perfusion pressure.
Correct Answer is B
Explanation
Choice A reason: The cells of the myocardium become hypertrophic in response to increased workload or pressure, not as a result of myocardial infarction. Hypertrophy is an adaptive mechanism that can lead to impaired ventricular function over time.
Choice B reason: The resulting hypoxia leads to ischemic injury and myocardial cell death. This is the main cause of impaired ventricular function after a myocardial infarction. The loss of viable myocardial tissue reduces the contractility and pumping ability of the heart.
Choice C reason: There is a temporary alteration in electrolyte balance that can be corrected. This is not the primary cause of impaired ventricular function after a myocardial infarction. Electrolyte imbalance can occur due to fluid loss, renal impairment, or medication side effects, but it can be managed with appropriate interventions.
Choice D reason: There is too much pressure on the heart and the ventricles begin to dysfunction. This is not the direct cause of impaired ventricular function after a myocardial infarction. Increased pressure on the heart can result from hypertension, valvular disease, or pulmonary embolism, but it is not related to myocardial ischemia or necrosis.
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