The forward effects in a patient with left ventricular heart failure include (Select all that apply)

Choice A reason: The cells of the myocardium become hypertrophic in response to increased workload or pressure, not as a result of myocardial infarction. Hypertrophy is an adaptive mechanism that can lead to impaired ventricular function over time.

Choice B reason: The resulting hypoxia leads to ischemic injury and myocardial cell death. This is the main cause of impaired ventricular function after a myocardial infarction. The loss of viable myocardial tissue reduces the contractility and pumping ability of the heart.

Choice C reason: There is a temporary alteration in electrolyte balance that can be corrected. This is not the primary cause of impaired ventricular function after a myocardial infarction. Electrolyte imbalance can occur due to fluid loss, renal impairment, or medication side effects, but it can be managed with appropriate interventions.

Choice D reason: There is too much pressure on the heart and the ventricles begin to dysfunction. This is not the direct cause of impaired ventricular function after a myocardial infarction. Increased pressure on the heart can result from hypertension, valvular disease, or pulmonary embolism, but it is not related to myocardial ischemia or necrosis.

Choice A reason: Infection in the blood is not empyema. It is sepsis, which is a life-threatening condition that occurs when the body's response to infection causes tissue damage, organ failure, or death.

Choice B reason: Infection in the pleural space is empyema. It is a collection of pus in the space between the lungs and the chest wall, usually caused by a bacterial infection that spreads from the lungs or the chest cavity.

Choice C reason: Exudative bronchitis is not empyema. It is a type of bronchitis that involves inflammation and excess mucus production in the bronchi, the airways that carry air to the lungs.

Choice D reason: Infection localized in the lung is not empyema. It is pneumonia, which is an inflammation of the lung tissue, usually caused by a viral or bacterial infection that affects the air sacs or the interstitial tissue.