Increased gastric motility and digestion are controlled by which of the following?
The parasympathetic nervous system
The limbic system
The central nervous system
The sympathetic nervous system
The Correct Answer is A
A) The parasympathetic nervous system:
This is the correct answer. The parasympathetic nervous system is responsible for regulating rest and digestion. It controls activities such as increased gastric motility, secretion of digestive enzymes, and relaxation of sphincters in the gastrointestinal tract. Activation of the parasympathetic nervous system promotes digestion and absorption of nutrients by increasing gastrointestinal activity.
B) The limbic system:
The limbic system is primarily involved in emotions, behavior, and long-term memory formation. While emotions can influence gastrointestinal function, including appetite and digestion, the limbic system itself does not directly control gastric motility and digestion.
C) The central nervous system:
The central nervous system includes the brain and spinal cord and plays a vital role in integrating and coordinating all body activities. While it indirectly influences gastrointestinal function through autonomic nervous system control, it is not the primary regulator of gastric motility and digestion.
D) The sympathetic nervous system:
The sympathetic nervous system is responsible for the body's fight or flight response, which involves activities such as increasing heart rate, dilating airways, and redirecting blood flow away from the digestive organs to skeletal muscles during times of stress or arousal. It typically inhibits digestive processes, including gastric motility, to conserve energy for immediate survival needs.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
This is the correct response. ACE inhibitors are known to cause a persistent, dry, and irritating cough in some individuals. This adverse effect occurs due to the accumulation of bradykinin and other substances in the lungs, leading to increased sensitivity of the cough reflex. The cough typically resolves upon discontinuation of the ACE inhibitor.
B) Respiratory depression:
Respiratory depression is not a common side effect of ACE inhibitors. ACE inhibitors do not directly affect respiratory drive or function in the central nervous system. Therefore, this option is incorrect.
C) Spontaneous pneumothorax:
Spontaneous pneumothorax is not a typical adverse effect associated with ACE inhibitor use. ACE inhibitors do not directly cause the development of pneumothorax, which is characterized by the presence of air in the pleural space. Therefore, this option is incorrect.
D) Pneumonia:
While ACE inhibitors can increase the risk of respiratory infections due to their effect on the immune system, pneumonia is not a specific adverse effect associated with ACE inhibitor use. Pneumonia is typically caused by infectious agents such as bacteria, viruses, or fungi, rather than being a direct effect of ACE inhibitors. Therefore, this option is incorrect.”
Correct Answer is A
Explanation
A) STAT administration of atropine:
This is the correct answer. Atropine is a cholinergic antagonist that can increase heart rate by blocking the action of acetylcholine on cardiac muscarinic receptors. In cases of severe bradycardia, especially if associated with symptoms such as dizziness, syncope, or hypotension, atropine is often administered to increase heart rate and improve cardiac output. The dose of atropine and frequency of administration depend on the severity of bradycardia and the clinical response.
B) Administration of activated charcoal:
Activated charcoal is used in cases of overdose or poisoning to absorb ingested toxins and prevent their absorption into the bloodstream. However, in this scenario, where the primary concern is bradycardia resulting from cholinesterase inhibitor (donepezil) toxicity, activated charcoal would not be effective in reversing the bradycardic effects of the medication.
C) Hemodialysis:
Hemodialysis is a renal replacement therapy used to remove toxins and waste products from the blood in individuals with kidney failure. While hemodialysis may be indicated in cases of severe drug overdose or poisoning to enhance toxin elimination, it is not typically used as a first-line intervention for bradycardia associated with cholinesterase inhibitor toxicity.
D) Intravenous administration of pseudoephedrine:
Pseudoephedrine is a sympathomimetic drug that acts as a vasoconstrictor and can increase heart rate and blood pressure. While it may be used to treat bradycardia in some cases, such as severe symptomatic bradycardia unresponsive to atropine, it is not the first-line treatment for cholinesterase inhibitor toxicity-induced bradycardia. Atropine is preferred due to its direct antagonism of muscarinic receptors in the heart.
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