In general, therapeutic interventions for a patient with osteoporosis will probably include drugs that

decrease osteoclastic activity

increase the kidneys tubular secretion of calcium into the urine.

increase resorption of calcium

suppress calcitonin

Answer and Explanation

The Correct Answer is A

A. Decrease osteoclastic activity: Osteoporosis treatments often focus on reducing osteoclastic activity, which is responsible for bone resorption, thereby helping to maintain or increase bone density.

B. Increase the kidneys' tubular secretion of calcium into the urine: This would decrease calcium levels in the body, which is not desired in osteoporosis treatment, as it would worsen bone density.

C. Increase resorption of calcium: Increasing bone resorption would exacerbate osteoporosis by weakening the bones further.

D. Suppress calcitonin: Calcitonin helps to inhibit bone resorption, so suppressing it would be counterproductive in treating osteoporosis.

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Correct Answer is C

Explanation

A. Moon face, buffalo hump, and hyperglycemia: These manifestations are associated with Cushing's disease, not Addison's disease.

B. Hirsutism, fever, and irritability: These symptoms are not characteristic of Addison's disease.

C. Anorexia, fatigue, and hypotension: Addison's disease often presents with symptoms like anorexia, fatigue, hypotension, and hyperpigmentation of the skin.

D. Tachycardia, exophthalmos, and goiter: These symptoms are associated with hyperthyroidism, not Addison's disease.

A. A sudden, explosive, disorderly charge of neurons causes a transient aberration in brain function B. Loss of the myelin sheath surrounding peripheral nerves causes asymmetric weakness C. Destruction of acetylcholine receptors causes muscle weakness with prolonged activity. D. A bacterial inflammatory illness that causes headache and photophobia.

Correct Answer is C

Explanation

A. A sudden, explosive, disorderly charge of neurons causes a transient aberration in brain function: This describes the pathophysiology of seizures, not myasthenia gravis.

B. Loss of the myelin sheath surrounding peripheral nerves causes asymmetric weakness: This describes multiple sclerosis, not myasthenia gravis.

C. Destruction of acetylcholine receptors causes muscle weakness with prolonged activity: Myasthenia gravis is an autoimmune disease where antibodies attack acetylcholine receptors at the neuromuscular junction, leading to muscle weakness, especially after repeated use.

D. A bacterial inflammatory illness that causes headache and photophobia: This describes meningitis, not myasthenia gravis.

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In general, therapeutic interventions for a patient with osteoporosis will probably include drugs that

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