In general, therapeutic interventions for a patient with osteoporosis will probably include drugs that
decrease osteoclastic activity
increase the kidneys tubular secretion of calcium into the urine.
increase resorption of calcium
suppress calcitonin
The Correct Answer is A
A. Decrease osteoclastic activity: Osteoporosis treatments often focus on reducing osteoclastic activity, which is responsible for bone resorption, thereby helping to maintain or increase bone density.
B. Increase the kidneys' tubular secretion of calcium into the urine: This would decrease calcium levels in the body, which is not desired in osteoporosis treatment, as it would worsen bone density.
C. Increase resorption of calcium: Increasing bone resorption would exacerbate osteoporosis by weakening the bones further.
D. Suppress calcitonin: Calcitonin helps to inhibit bone resorption, so suppressing it would be counterproductive in treating osteoporosis.
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Related Questions
Correct Answer is C
Explanation
A. Administer some insulin: Insulin is not appropriate unless there is evidence of hyperglycemia, which is not suggested by the symptoms described.
B. Prepare the patient to receive a blood transfusion: The symptoms described are not indicative of a need for a blood transfusion but rather suggest adrenal insufficiency.
C. Administer IV glucose: Addisonian crisis can be triggered by stress and dehydration, leading to low blood sugar and symptoms such as weakness and confusion. Administering IV glucose can help manage hypoglycemia and provide immediate support.
D. Collect some urine and assess for glucosuria: The symptoms are more acute and related to adrenal insufficiency rather than glucosuria.
Correct Answer is D
Explanation
A. Hyperproteinemia and increased drug effect: In cirrhosis, hypoproteinemia (low protein levels) occurs due to decreased protein synthesis by the liver, and drug metabolism is often impaired, leading to increased drug effects, but hyperproteinemia is not a typical finding.
B. Hyperkalemia and fluid retention: While fluid retention is common due to hypoalbuminemia and portal hypertension, hyperkalemia is not a direct consequence of hepatocyte dysfunction.
C. Hypercortisolism and increased infection risk: Hypercortisolism is not typically associated with cirrhosis. However, increased infection risk is common due to compromised immune function.
D. An elevated blood glucose and ammonia level: In cirrhosis, the liver's ability to metabolize ammonia is impaired, leading to elevated levels. Additionally, impaired glucose metabolism can result in hyperglycemia.
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