An adolescent boy is admitted to the emergency department following a bee sting. He arrives with a body rash and 30 minutes later becomes short of breath. The nurse obtains vital signs with a blood pressure of 90/52 mm Hg, heart rate 130 beats/minute, and respiratory rate 40 breaths/minute. The client is exhibiting clinical manifestations of which type of immune reaction?
IgE-mediated response
Cell-mediated hypersensitivity
Autoimmune response
Type II hypersensitivity
The Correct Answer is A
Choice A reason: An IgE-mediated (Type I) hypersensitivity reaction, like anaphylaxis, occurs post-bee sting, with rapid onset of rash, shortness of breath, hypotension, and tachycardia. IgE antibodies trigger mast cell degranulation, releasing histamine, causing vasodilation, bronchoconstriction, and systemic symptoms, matching the client’s acute presentation.
Choice B reason: Cell-mediated (Type IV) hypersensitivity involves T-cells, causing delayed reactions like contact dermatitis, not rapid systemic symptoms. Bee sting reactions are immediate, driven by IgE, not T-cells. Rash, hypotension, and respiratory distress indicate anaphylaxis, not a delayed cell-mediated response.
Choice C reason: Autoimmune responses involve self-directed antibodies, as in lupus, not triggered by external allergens like bee stings. The client’s acute rash, hypotension, and respiratory distress suggest an IgE-mediated anaphylactic reaction, not an autoimmune process, which is unrelated to acute allergen exposure.
Choice D reason: Type II hypersensitivity involves antibody-mediated cytotoxicity, as in transfusion reactions, not allergen-induced systemic symptoms. Bee sting reactions are IgE-driven, causing immediate anaphylaxis with rash and hypotension, not cytotoxic damage, making Type II inappropriate for the client’s acute presentation.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice A reason: Chest x-ray may show nonspecific findings in pulmonary embolism but is not diagnostic. D-dimer, a fibrin degradation product, is highly sensitive for detecting thrombosis, guiding further imaging like CT pulmonary angiography, making it the priority test in suspected cases.
Choice B reason: Electrocardiogram may show tachycardia or right heart strain in pulmonary embolism but is nonspecific. D-dimer is a sensitive screening test, indicating clot presence, which prompts definitive imaging, making it the preferred initial diagnostic step in suspected pulmonary embolism.
Choice C reason: D-dimer blood test is the most sensitive screening tool for pulmonary embolism, detecting fibrin degradation from clots. Elevated levels prompt CT pulmonary angiography for confirmation, guiding urgent treatment to prevent further embolization, making it the critical test to anticipate.
Choice D reason: Arterial blood gas may reveal hypoxemia in pulmonary embolism but is nonspecific, as other conditions cause similar findings. D-dimer is more diagnostic, identifying clot presence, guiding imaging, and ensuring timely intervention, making it the priority test in suspected cases.
Correct Answer is B
Explanation
Choice A reason: Promoting effective swallowing is relevant for dysphagia but not the primary goal for sliding hiatal hernia, where the stomach protrudes into the thorax, causing reflux. Preventing acid reflux into the esophagus is critical to reduce irritation and complications like esophagitis, making swallowing secondary.
Choice B reason: Preventing esophageal reflux is the primary goal for sliding hiatal hernia, as the stomach’s protrusion through the diaphragm allows gastric acid to reflux, causing heartburn and esophageal damage. Nursing actions like elevating the head of the bed and avoiding trigger foods minimize reflux, protecting the esophagus.
Choice C reason: Maintaining intact oral mucosa is unrelated to hiatal hernia, which affects the gastroesophageal junction. Reflux causes esophageal, not oral, irritation. The focus is on preventing acid reflux to protect the esophagus, making oral mucosa maintenance irrelevant to the condition’s pathophysiology.
Choice D reason: Increasing intestinal peristalsis is not a goal for hiatal hernia, which involves gastroesophageal reflux, not intestinal motility. Enhanced peristalsis may worsen reflux by increasing intra-abdominal pressure. Preventing reflux is prioritized to manage symptoms and prevent esophageal complications.
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