A nurse is reviewing a client's laboratory results. The client's ABGS are as follows: pH 7.6, HCO3 24 mEq/L, PaCO2 30 mm Hg, PaO2 90 mm Hg. Which of the following acid-base imbalances should the nurse identify the client is experiencing?
Metabolic alkalosis
Respiratory acidosis
Respiratory alkalosis
Metabolic acidosis
The Correct Answer is C
A) Metabolic alkalosis: Metabolic alkalosis is characterized by a high pH and elevated bicarbonate (HCO3). In this scenario, the pH is elevated at 7.6, which supports alkalosis, but the HCO3 level is normal at 24 mEq/L. The PaCO2 is slightly low, which is not typical for metabolic alkalosis, as it would usually show an elevated HCO3 with a compensatory respiratory alkalosis.
B) Respiratory acidosis: Respiratory acidosis would present with a low pH and an elevated PaCO2. In this case, the pH is high at 7.6, indicating alkalosis, and the PaCO2 is also low at 30 mm Hg, which is inconsistent with respiratory acidosis.
C) Respiratory alkalosis: Respiratory alkalosis is indicated by a high pH with a low PaCO2. Here, the pH is elevated at 7.6, and the PaCO2 is decreased at 30 mm Hg, which fits the profile of respiratory alkalosis. The normal HCO3 level suggests that the bicarbonate is not compensating, supporting a primary respiratory alkalosis.
D) Metabolic acidosis: Metabolic acidosis is characterized by a low pH and a low HCO3. In this scenario, the pH is elevated at 7.6, and the HCO3 level is normal at 24 mEq/L, which does not align with metabolic acidosis. The PaCO2 is also low, which is not typical for metabolic acidosis, as it would usually have a normal or high PaCO2.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A) Decrease folic acid intake: Decreasing folic acid intake is not advisable for clients with malabsorption syndrome. In fact, clients with malabsorption often require increased folic acid due to poor absorption of nutrients, especially in conditions like celiac disease or other causes of malabsorption.
B) Increase lactose intake: Increasing lactose intake is not appropriate for clients with malabsorption syndrome, particularly if they have lactose intolerance. Lactose can exacerbate symptoms such as diarrhea and steatorrhea in these clients.
C) Provide a gluten-free diet: A gluten-free diet is crucial for clients with celiac disease, which is a type of malabsorption syndrome. However, if the malabsorption syndrome is due to another cause, a gluten-free diet might not address the issue. The focus should be on managing fat intake to reduce steatorrhea.
D) Provide a low-fat diet: A low-fat diet is the most appropriate intervention for managing severe steatorrhea, which is the presence of excess fat in the stool. Reducing fat intake helps to minimize fat malabsorption and the associated symptoms, providing relief from steatorrhea.
Correct Answer is B
Explanation
A) HDL 65 mg/dL: High-density lipoprotein (HDL) levels of 65 mg/dL are considered protective against heart disease. Higher HDL levels are generally associated with a lower risk of cardiovascular disease, so this result is not a concern.
B) LDL 120 mg/dL: Low-density lipoprotein (LDL) levels of 120 mg/dL are elevated for individuals at increased risk of cardiovascular disease, especially with a family history of hyperlipidemia and cardiac disease. An LDL level above 100 mg/dL is a risk factor for heart disease, making this result important to report to the provider for potential intervention.
C) Cholesterol 195 mg/dL: A total cholesterol level of 195 mg/dL is slightly above the recommended level of less than 200 mg/dL but may not be immediately alarming unless the client has other risk factors. However, this value alone is not as critical as LDL levels in assessing risk.
D) Triglycerides 175 mg/dL: Triglyceride levels of 175 mg/dL are slightly elevated, as normal levels are typically less than 150 mg/dL. While elevated triglycerides are a risk factor for cardiovascular disease, this level is less critical compared to elevated LDL levels in the context of the client’s family history.
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