A nurse in a clinic is caring for an adolescent client who is at 24 weeks of gestation and showing signs of preeclampsia. Which of the following findings should the nurse expect?
Decreased BUN
Increased protein in urine
Increased platelet count
Decreased serum uric acid
The Correct Answer is B
Choice A reason: Decreased BUN is not typical in preeclampsia, where renal impairment often elevates BUN due to reduced glomerular filtration. Normal or increased BUN is expected, so this finding does not align with preeclampsia’s pathophysiology, making it an incorrect expectation.
Choice B reason: Increased protein in urine (proteinuria) is a hallmark of preeclampsia, resulting from glomerular damage due to hypertension and endothelial dysfunction. This diagnostic criterion, often >300 mg/24 hours, is critical for identifying preeclampsia, making it the correct finding the nurse should expect.
Choice C reason: Increased platelet count is not associated with preeclampsia, which often causes thrombocytopenia due to endothelial activation and platelet consumption. A decreased count (<100,000/mm³) is more likely, making this finding incorrect for preeclampsia’s clinical presentation.
Choice D reason: Decreased serum uric acid is not expected in preeclampsia, where elevated uric acid occurs due to reduced renal clearance from glomerular dysfunction. Increased levels are a marker, so this finding is opposite to preeclampsia’s effects, making it incorrect.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: Calcium gluconate IV reverses magnesium sulfate toxicity, which causes respiratory depression or arrhythmias due to excessive magnesium. Calcium restores neuromuscular and cardiac function by competing with magnesium, preventing life-threatening complications like respiratory arrest in preeclampsia management.
Choice B reason: Positioning supine is inappropriate, as it does not address magnesium toxicity and may worsen respiration in preeclampsia. Semi-Fowler’s position optimizes breathing, while toxicity requires pharmacological reversal with calcium gluconate, not positional changes, to manage life-threatening symptoms effectively.
Choice C reason: IV dextrose is irrelevant for magnesium toxicity, which affects neuromuscular function, not glucose levels. Dextrose treats hypoglycemia, not applicable here. Magnesium overdose requires calcium to counteract effects, making dextrose an ineffective intervention in preeclampsia-related toxicity management.
Choice D reason: Methylergonovine, a uterotonic, is contraindicated in preeclampsia, as it increases blood pressure, risking hypertensive crisis. It treats postpartum hemorrhage, not magnesium toxicity, which requires calcium gluconate to reverse neuromuscular depression, ensuring safety in preeclampsia management.
Correct Answer is B
Explanation
Choice A reason: Cardiogenic shock involves reduced cardiac output, typically with low PAWP due to decreased preload from poor heart function. Elevated PAWP reflects left atrial pressure buildup, not characteristic of cardiogenic shock alone, which is distinct from heart failure, requiring specific hemodynamic management.
Choice B reason: Elevated PAWP indicates left ventricular failure, where the heart cannot pump blood effectively, causing pulmonary congestion. This increases left atrial pressure, leading to pulmonary edema, a hallmark of heart failure. Diuretics and inotropes are needed to reduce fluid overload and improve cardiac function.
Choice C reason: Hypotension is a symptom, not a direct complication of elevated PAWP. It may occur in heart failure due to reduced cardiac output, but elevated PAWP specifically signals left heart dysfunction, not hypotension itself, which is a systemic response rather than a primary cardiac issue.
Choice D reason: Hypovolemia reduces blood volume, lowering PAWP due to decreased preload. Elevated PAWP suggests fluid overload or left ventricular dysfunction, not hypovolemia, which presents with low central venous pressure and dehydration signs, requiring fluid resuscitation rather than management of heart failure.
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