A client with a medical diagnosis of acute respiratory distress syndrome (ARDS) is being placed in the prone position. The nurse explains to family members that, "This will help improve ventilation by:

A. Speak slow and loud so that the client can understand speech:

While speaking slowly and clearly may help with communication in some patients with ALS, this approach is less effective as the disease progresses. As ALS advances, speech muscles become affected, and the client may be unable to speak at all, making this method inappropriate for end-stage ALS. Simply speaking louder does not address the communication needs of a client who can no longer use their voice.

B. Encourage the client to write questions on a white erase board:

While writing on a whiteboard may be an effective communication strategy in the early stages of ALS when the client still has adequate hand function, this becomes increasingly difficult as muscle weakness progresses. By the end-stage, clients may lose the ability to hold a pen or write legibly, making this method less viable as the disease advances. It is not the best option for clients with significant motor impairment.

C. Use pre-arranged eye signals because eye muscles remain intact: In clients with end-stage amyotrophic lateral sclerosis (ALS), muscle weakness and loss of voluntary control progressively affect motor functions, including the ability to speak, write, and move. However, the eye muscles are typically preserved until the later stages of the disease, allowing patients to communicate through eye movements. Pre-arranged eye signals, such as blinking for "yes" or "no" or using a system of eye movements to select letters or words, are effective ways to facilitate communication with clients who can no longer speak or write. This method respects the client's remaining abilities and allows for more effective communication, especially as the disease progresses to its final stages.

D. Have the client squeeze the nurse's hand when intubated and on the ventilator:

When a client is intubated and on a ventilator, their ability to squeeze the nurse's hand may be limited due to the sedation and ventilator settings, and they may not have sufficient motor control to respond consistently. Additionally, when intubated, respiratory effort is controlled by the ventilator, so relying on hand squeezing would not be an effective or reliable method of communication. Furthermore, ALS patients in the later stages may not have enough muscle control for this method to be practical.

A) Severe left-sided heart failure and resultant pulmonary edema:
While pulmonary edema due to left-sided heart failure can lead to respiratory distress and hypoxemia, it is not characteristic of ARDS. ARDS is a form of non-cardiogenic pulmonary edema, meaning it is not caused by heart failure. In contrast, pulmonary edema from heart failure is typically related to increased pressure in the pulmonary circulation. Therefore, while this client is at risk for respiratory issues, the cause of their pulmonary edema is distinct from the pathology seen in ARDS.

B) Acute renal failure associated with pyelonephritis:
Acute renal failure from pyelonephritis can lead to various complications, including electrolyte imbalances and fluid overload, which may affect respiratory function. However, renal failure by itself is not a direct cause of ARDS. ARDS is typically associated with an inflammatory response to injury or infection in the lungs, not specifically renal issues. While it’s important to monitor for pulmonary complications in critically ill clients, this situation does not directly suggest ARDS.

C) A traumatic brain injury with accompanying spinal cord injury:
Traumatic brain injury (TBI) with spinal cord injury can lead to respiratory compromise, particularly due to neurological impairment affecting the respiratory muscles or the brain's ability to control breathing. However, ARDS is not the most direct consequence of these injuries. ARDS is primarily caused by acute lung injury from direct or indirect insults to the lungs, such as trauma, pneumonia, or sepsis. Although this combination of injuries may cause respiratory distress, it is not a typical cause of ARDS unless there is another underlying lung injury.

D) Hypoxemia, refractory to oxygen therapy:
This is the hallmark sign of ARDS. ARDS is characterized by the development of acute hypoxemia that is resistant to high levels of supplemental oxygen therapy. This refractory hypoxemia is due to widespread inflammation and damage to the alveolar-capillary membrane, leading to impaired gas exchange. In ARDS, the lungs become less compliant, and the ability to oxygenate blood is significantly reduced, even with mechanical ventilation and high levels of oxygen. Therefore, a critically ill client with hypoxemia that does not improve with oxygen therapy would raise suspicion for the development of ARDS.