The nurse is caring for a client with a new diagnosis of anemia. Which dietary recommendation should the nurse provide?
Increase iron-rich foods
Limit vitamin C intake
Restrict green leafy vegetables
Avoid whole grains
The Correct Answer is A
Choice A reason: Increasing iron-rich foods (e.g., red meat, spinach) corrects anemia by boosting hemoglobin synthesis, as iron is essential for red blood cell production. This addresses the underlying deficiency, improving oxygen transport, making it the priority dietary recommendation for anemia management.
Choice B reason: Limiting vitamin C is incorrect, as it enhances iron absorption, aiding anemia correction. Iron-rich foods are the priority, as they directly supply the deficient nutrient, improving hemoglobin levels, making vitamin C restriction counterproductive to effective anemia treatment.
Choice C reason: Restricting green leafy vegetables, which are iron-rich, worsens anemia. These foods (e.g., spinach) provide dietary iron, essential for hemoglobin synthesis. Increasing iron intake is the priority, as it corrects the deficiency, making vegetable restriction inappropriate for anemia.
Choice D reason: Avoiding whole grains is irrelevant, as they provide nutrients supporting overall health. Iron-rich foods directly address anemia by supplying iron for red blood cell production, making them the priority dietary recommendation over grain restrictions, which do not impact anemia correction.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A reason: Anorexia is common in hepatitis due to systemic inflammation but is nonspecific, occurring in many conditions. Clay-colored stools directly indicate impaired bile excretion from liver dysfunction, a hallmark of hepatitis, making it a more diagnostic finding.
Choice B reason: Clay-colored stools are highly indicative of hepatitis, as liver inflammation impairs bile production or excretion, reducing bilirubin in feces. This causes pale stools, reflecting hepatic dysfunction, making it a key finding to confirm hepatitis over nonspecific symptoms like anorexia.
Choice C reason: Brown, foamy urine is not typical of hepatitis. Dark urine from bilirubinuria may occur, but foamy urine suggests proteinuria, unrelated to liver dysfunction. Clay-colored stools are more specific, indicating bile flow obstruction, a direct sign of hepatitis pathology.
Choice D reason: Hematemesis indicates gastrointestinal bleeding, not a primary hepatitis feature. While advanced liver disease may cause variceal bleeding, clay-colored stools are an earlier, more specific sign of hepatitis-related bile impairment, making them the priority finding in acute cases.
Correct Answer is B
Explanation
Choice A reason: HIV does not primarily cause a deficiency in antibody production. B-cells produce antibodies, but HIV targets CD4 T-cells, impairing their ability to activate B-cells. This indirectly reduces antibody effectiveness, but the primary mechanism is T-cell destruction, not a direct antibody production deficit, making this incorrect.
Choice B reason: HIV infects and destroys helper T-cells (CD4 cells), critical for coordinating immune responses. By reducing CD4 cell counts, HIV impairs activation of B-cells and cytotoxic T-cells, leading to immune suppression. This is the primary mechanism of AIDS-related immune deficiency, making it the correct explanation for HIV pathology.
Choice C reason: Proliferation of suppressor T-cells (regulatory T-cells) is not a primary HIV mechanism. HIV depletes CD4 cells, not suppressor T-cells, which modulate immune responses. While immune dysregulation occurs, the hallmark is CD4 destruction, not suppressor T-cell proliferation, making this an inaccurate description of HIV’s action.
Choice D reason: HIV does not increase B-lymphocyte numbers. It impairs B-cell function indirectly by destroying CD4 cells, which are needed to activate B-cells for antibody production. B-cell hyperactivity may occur in early HIV, but the primary immune suppression results from CD4 cell loss, not B-cell proliferation.
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