A client is admitted with a closed head injury sustained in a motor vehicle accident.
The nurse notes that the client's intracranial pressure (ICP) shows an upward trend. Which action should the nurse prioritize?
Administer 100 mg of pentobarbital IV as ordered.
Administer 1 g Mannitol IV as ordered.
Increase the ventilator respiratory rate to 20 breaths/minute.
Reposition the client to avoid neck flexion.
The Correct Answer is B
Managing increased intracranial pressure requires applying knowledge of osmotic therapy, cerebral perfusion, and neurological positioning. Nurses must prioritize interventions that rapidly reduce cerebral edema while maintaining physiological stability and preventing herniation in patients with traumatic brain injuries or intracranial hypertension.
Choice A rationale
Pentobarbital is a barbiturate used for medically induced comas to reduce cerebral metabolic demand. While effective for refractory intracranial hypertension, it is typically a secondary or tertiary intervention rather than the immediate priority for an upward trend in pressure.
Choice B rationale
Mannitol is an osmotic diuretic that creates an osmotic gradient, pulling fluid from the cerebral interstitial space into the vascular compartment. This rapidly reduces brain volume and intracranial pressure, making it the first-line pharmacological priority for acute elevations.
Choice C rationale
Hyperventilation decreases PaCO2, causing cerebral vasoconstriction and reduced blood volume. However, excessive hyperventilation can cause cerebral ischemia. Increasing the rate to 20 should only be done under specific medical direction and is not the primary nursing priority here.
Choice D rationale
Proper positioning, such as head of bed elevation and avoiding neck flexion, facilitates venous drainage from the brain. While important, it is a supportive measure and less effective than osmotic diuretics for treating an acute, upward trend.
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Related Questions
Correct Answer is A
Explanation
This scenario requires understanding acid-base balance and renal pathophysiology. Acute kidney injury impairs the kidneys ability to excrete hydrogen ions and regenerate bicarbonate. Identifying metabolic acidosis with partial respiratory compensation is necessary based on the interpretation of arterial blood gas values.
Choice A rationale
Metabolic acidosis occurs in kidney injury due to decreased acid excretion. The pH is low at 7.26 (7.35 to 7.45) and bicarbonate is low at 14 (22 to 28), while the PaCO2 of 30 shows compensatory hyperventilation.
Choice B rationale
A pH of 7.26 indicates acidosis, but the normal bicarbonate level of 24 and elevated PaCO2 of 46 indicate respiratory acidosis. This occurs with hypoventilation and carbon dioxide retention rather than the metabolic issues seen in kidney failure.
Choice C rationale
The pH of 7.49 indicates alkalosis. A high bicarbonate of 30 with a normal PaCO2 of 40 represents uncompensated metabolic alkalosis. This is typical of excessive acid loss or base gain, which is opposite of kidney failure.
Choice D rationale
The pH of 7.49 and low PaCO2 of 30 suggest respiratory alkalosis. Bicarbonate is normal at 24. This state results from hyperventilation and excessive carbon dioxide loss, which does not characterize the pathophysiology of acute kidney injury.
Correct Answer is []
Explanation
Rationale for correct condition hemorrhagic stroke is strongly indicated by the "worst headache of my life," sudden aphasia, and left-sided weakness. an inr of 4.9 is critically high, significantly increasing bleeding risk. negative kernig and brudzinski signs help rule out meningitis despite the fever history. the presence of photophobia and vision loss further supports an intracranial event.
Rationale for correct actions a stat ct brain is the gold standard to differentiate between ischemic and hemorrhagic events and identify intracranial bleeding. a lumbar puncture may be indicated if the ct is negative but clinical suspicion of subarachnoid hemorrhage remains high. these actions are time-sensitive to prevent secondary brain injury from increased intracranial pressure. normal inr for a client not on anticoagulants is 0.8-1.1. the nurse must prioritize diagnostic imaging to guide surgical or medical intervention. these steps address the acute neurological deficit and high-intensity headache.
Rationale for correct parameters blood pressure must be monitored strictly as hypertension can exacerbate intracranial bleeding and worsen stroke outcomes. the client's blood pressure rose from 119/85 to 159/83 mm hg, indicating a need for tight control. monitoring pt/inr is vital due to the supratherapeutic level of 4.9, which necessitates reversal agents like vitamin k or fresh frozen plasma. normal prothrombin time is typically 11-13.5 seconds. managing these parameters prevents further hematoma expansion. frequent neurological checks are also implicit in monitoring these physiological markers.
Rationale for incorrect conditions migraine headache does not typically present with sudden aphasia, hemiparesis, or an inr of 4.9. trigeminal neuralgia is characterized by stabbing facial pain, which the client explicitly denied. meningitis is unlikely given the negative kernig and brudzinski signs and the sudden, focal neurological deficits. while the client had a fever, the clinical picture is dominated by vascular and neurological collapse.
Rationale for incorrect actions administering sumatriptan is contraindicated in suspected stroke as it causes vasoconstriction and is intended for migraines. carbamazepine is the specific treatment for trigeminal neuralgia, which does not match this client's presentation. providing ibuprofen or other anticoagulants would be dangerous given the already elevated inr of 4.9.
Rationale for incorrect parameters a headache diary for food triggers is a long-term management tool for migraine headache, not an acute stroke. temperature monitoring is less critical than hemodynamics in the immediate management of a suspected hemorrhage. while the wbc is slightly elevated, it is expected with a uti and is less urgent than the neurological crisis.
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