A client diagnosed with diabetes mellitus is admitted to the emergency department (ED) and the cardiac monitor shows multifocal premature ventricular contractions (PVCs). Initial laboratory findings are as follows: The nurse recognizes that the client's PVCs are most likely caused by:
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Potassium 2.8 mEq/L |
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Sodium 133 mEq/L |
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Chloride 90 mEq/L |
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Glucose 200 mg/dl |
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Magnesium 2.5 mg/dL |
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Calcium 8.0 mg/dL |
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hypermagnesemia.
hypocalcemia.
hypokalemia.
hyperglycemia.
The Correct Answer is C
A. Hypermagnesemia: Hypermagnesemia (high magnesium levels) can cause bradycardia, hypotension, and prolonged PR and QRS intervals. While it can affect cardiac rhythm, it's less commonly associated with PVCs. The client's magnesium level of 2.5 mg/dL is within the normal range (1.5-2.5 mg/dL).
B. Hypocalcemia: While the calcium level of 8.0 mg/dL is slightly low (normal: ~8.5–10.5 mg/dL), mild hypocalcemia is less commonly associated with PVCs compared to hypokalemia. It can affect cardiac contractility but is not the most likely cause of these arrhythmias.
C. Hypokalemia: The potassium level is 2.8 mEq/L, which is significantly below normal (normal: 3.5–5.0 mEq/L). Potassium is a crucial electrolyte for maintaining normal cardiac electrical activity. Hypokalemia increases myocardial excitability and can lead to various cardiac dysrhythmias and a known cause of ventricular irritability, including multifocal PVCs, and increases the risk of life-threatening arrhythmias in clients with cardiac or metabolic conditions.
D. Hyperglycemia: The glucose level of 200 mg/dL is elevated but not severely high. While it reflects poor glycemic control, it is not directly linked to the occurrence of PVCs. Electrolyte imbalances, particularly low potassium, are more arrhythmogenic.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. Increase in systolic blood pressure 10 mmHg above client's norm: A mild increase in systolic blood pressure is not a specific indicator of mitral stenosis progression. Blood pressure fluctuations may occur due to various factors and are not typically used as a primary marker of worsening valve disease.
B. reports dyspnea after walking up 2 flights of stairs: Exertional dyspnea is a common early symptom of mitral stenosis due to reduced left atrial emptying and pulmonary congestion. However, this does not necessarily indicate worsening unless the symptom becomes more severe or occurs with minimal exertion.
C. Jugular vein distention and +3 peripheral edema: These are signs of right-sided heart failure, which may result from long-standing mitral stenosis leading to pulmonary hypertension and subsequent right ventricular hypertrophy and failure. This indicates disease progression and worsening cardiac function.
D. Complaints of epigastric pain after eating a large meal: Epigastric discomfort is not a typical symptom of mitral stenosis and is more likely related to gastrointestinal issues. It does not reflect the status or progression of the cardiac condition.
Correct Answer is D
Explanation
A. Maintain the infusion because the client had a cardiac arrest: While epinephrine is essential during cardiac arrest, continuing a high-dose infusion post-resuscitation without reassessment may lead to complications like tachycardia, hypertension, and increased myocardial oxygen demand.
B. Continue to monitor the client's rhythm closely: Ongoing monitoring is important, but it is a passive intervention. The heart rate of 120 bpm may reflect excessive adrenergic stimulation from epinephrine, and further action is needed to prevent deterioration.
C. Suggest that the client's medication be changed to norepinephrine: Norepinephrine is another vasopressor that has less of a beta-1 adrenergic effect compared to epinephrine. It is primarily used for hypotension and septic shock, not as a direct substitute for epinephrine post-cardiac arrest. Changing to another vasopressor without indication is not the best initial step.
D. Ask the physician if the dose can be decreased: A heart rate of 120 bpm may indicate that the epinephrine dose is too high, causing sympathetic overstimulation. Prolonged or excessive tachycardia increases myocardial oxygen demand, which can be detrimental, especially in a post-arrest heart. Decreasing the dose can help prevent arrhythmias or myocardial ischemia, making this the most appropriate and proactive action.
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