A nurse is preparing a client for an electroencephalogram (EEG). When the client asks what the purpose of the procedure is, what is the nurse's best response?

A. Respiratory acidosis:
Respiratory acidosis is characterized by an increased CO2 level (above 45 mmHg) due to hypoventilation or impaired gas exchange, leading to a decrease in pH (below 7.35). In this case, the CO2 level is 25 mmHg, which is lower than normal and suggests a loss of CO2, not an accumulation. Therefore, respiratory acidosis is not the correct interpretation of these ABG results.
B. Metabolic acidosis:
Metabolic acidosis occurs when the pH drops below 7.35 due to conditions like kidney failure, diarrhea, or diabetic ketoacidosis. This would typically be accompanied by a low bicarbonate (HCO3-) level (less than 22 mEq/L). However, in this case, the pH is 7.52 (alkalotic), and the HCO3- level is 24 mEq/L, which is within the normal range. Thus, metabolic acidosis is not the correct interpretation.

C. Respiratory alkalosis:
Respiratory alkalosis is characterized by a decrease in CO2 (less than 35 mmHg) due to hyperventilation, which causes an increase in pH (above 7.45). In this case, the pH is 7.52 (alkalotic), and the CO2 is 25 mmHg, which is lower than the normal range (35–45 mmHg). The bicarbonate (HCO3-) level is 24 mEq/L, which is normal and compensatory. This matches the pattern of respiratory alkalosis, where the body compensates with a normal bicarbonate level as CO2 decreases.

D. Metabolic alkalosis:
Metabolic alkalosis occurs when the pH rises above 7.45, often due to excessive vomiting, diuretic use, or antacid overuse, leading to an increase in bicarbonate (HCO3-) levels. While the pH is 7.52, indicating alkalosis, the bicarbonate (HCO3-) level is 24 mEq/L, which is within the normal range for bicarbonate. In metabolic alkalosis, you would typically see an elevated HCO3- level, which is not the case here. Therefore, metabolic alkalosis is not the correct interpretation of these ABG results.

A. 15-30g oral carbohydrates:
Oral carbohydrates are a first-line treatment for hypoglycemia in a conscious, alert patient who can safely swallow. However, since the patient is unresponsive, administering oral carbohydrates is not an appropriate option. The patient’s inability to swallow safely increases the risk of aspiration, making IV treatment the priority in this case.

B. 10% dextrose continuous IV infusion:
A 10% dextrose IV infusion can be used in the management of hypoglycemia, but in an acute, emergency setting where the patient is unresponsive and their blood glucose is critically low (30 mg/dL), a rapid-acting intervention is needed. A bolus dose of a concentrated solution, such as 50% dextrose, is more appropriate for quickly raising the blood glucose level in this situation, rather than a continuous infusion, which takes longer to achieve an effective increase in glucose.

C. Glucagon PO:
Glucagon is typically used for hypoglycemia in patients who are unconscious or unable to take oral glucose. However, glucagon is typically administered intramuscularly (IM) or subcutaneously (SQ), not orally (PO). Administering glucagon orally is ineffective, as it would not be absorbed by the body in the necessary manner to correct hypoglycemia. Therefore, this option is inappropriate.

D. 50% dextrose in water (50% DW) IV push:
When a patient is unresponsive and their blood glucose level is critically low (30 mg/dL), the priority treatment is an immediate, concentrated source of glucose. Administering 50% dextrose IV push is the most appropriate intervention in this scenario. It provides a rapid and effective increase in blood glucose levels, which is critical for reversing hypoglycemia in an emergency situation. This is the fastest and most direct approach to treating severe hypoglycemia in an unresponsive patient.